Forced expression of p21 in GPIIb-p21 transgenic mice induces abnormalities in the proliferation of erythroid and megakaryocyte progenitors and primitive hematopoietic cells. [electronic resource]
Producer: 20021212Description: 1263-72 p. digitalISSN:- 0301-472X
- Animals
- Blood Cell Count
- Cell Differentiation
- Cell Division
- Cell Lineage
- Colony-Forming Units Assay
- Cyclin-Dependent Kinase Inhibitor p21
- Cyclins -- biosynthesis
- DNA, Complementary -- genetics
- Erythroid Precursor Cells -- cytology
- Gene Expression Regulation
- Genes, Synthetic
- Hematopoiesis -- genetics
- Hematopoietic Stem Cells -- cytology
- Homeostasis
- Humans
- Megakaryocytes -- cytology
- Mice
- Mice, Transgenic
- Platelet Membrane Glycoprotein IIb -- genetics
- Promoter Regions, Genetic
- Recombinant Fusion Proteins -- physiology
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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