Stress-activated protein kinase/c-Jun N-terminal kinase (JNK) plays a part in endothelin-1-induced vascular endothelial growth factor synthesis in osteoblasts. [electronic resource]
Producer: 20030627Description: 417-23 p. digitalISSN:- 0730-2312
- Animals
- Anthracenes -- pharmacology
- Bone and Bones -- cytology
- Cells, Cultured
- Dose-Response Relationship, Drug
- Endothelial Growth Factors -- biosynthesis
- Endothelin-1 -- metabolism
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- Enzyme-Linked Immunosorbent Assay
- Imidazoles -- pharmacology
- Intercellular Signaling Peptides and Proteins -- biosynthesis
- JNK Mitogen-Activated Protein Kinases
- Lymphokines -- biosynthesis
- MAP Kinase Kinase 4
- MAP Kinase Signaling System
- Mice
- Mitogen-Activated Protein Kinase Kinases -- metabolism
- Mitogen-Activated Protein Kinases -- metabolism
- Osteoblasts -- enzymology
- Phosphorylation
- Pyridines -- pharmacology
- Time Factors
- Vascular Endothelial Growth Factor A
- Vascular Endothelial Growth Factors
- p38 Mitogen-Activated Protein Kinases
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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