Urinary tumor necrosis factor contributes to sodium retention and renal hypertrophy during diabetes. [electronic resource]
Producer: 20030108Description: F113-21 p. digitalISSN:- 1931-857X
- Albuminuria -- metabolism
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Antigens, CD -- metabolism
- Biomarkers
- Cell Separation
- Diabetes Mellitus, Experimental -- metabolism
- Diabetic Nephropathies -- metabolism
- Etanercept
- Hypertrophy
- Immunoglobulin G -- pharmacology
- Kidney -- metabolism
- Kidney Tubules, Distal -- cytology
- Male
- Organ Size
- Rats
- Rats, Sprague-Dawley
- Receptors, Tumor Necrosis Factor -- metabolism
- Receptors, Tumor Necrosis Factor, Type I
- Receptors, Tumor Necrosis Factor, Type II
- Sodium -- metabolism
- Tumor Necrosis Factor-alpha -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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