Physiological levels of interleukin-18 stimulate multiple neutrophil functions through p38 MAP kinase activation. [electronic resource]
Producer: 20020826Description: 401-9 p. digitalISSN:- 0741-5400
- Adult
- Calcium
- Calcium Signaling -- drug effects
- Cell Size
- Chelating Agents -- pharmacology
- Egtazic Acid -- analogs & derivatives
- Enzyme Inhibitors -- pharmacology
- Humans
- Imidazoles -- pharmacology
- Interleukin-18 -- pharmacology
- Lipopolysaccharides -- pharmacology
- MAP Kinase Signaling System -- drug effects
- Macrophage-1 Antigen -- biosynthesis
- Membrane Proteins -- metabolism
- Mitogen-Activated Protein Kinases -- antagonists & inhibitors
- NADPH Oxidases -- metabolism
- Neutrophils -- drug effects
- Phosphoproteins -- metabolism
- Protein Transport -- drug effects
- Pyridines -- pharmacology
- Receptors, Formyl Peptide
- Receptors, Immunologic -- biosynthesis
- Receptors, Peptide -- biosynthesis
- Respiratory Burst -- drug effects
- Up-Regulation -- drug effects
- p38 Mitogen-Activated Protein Kinases
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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