ICAM-1 and VCAM-1 mediate endotoxemic myocardial dysfunction independent of neutrophil accumulation. [electronic resource]
Producer: 20020815Description: R477-86 p. digitalISSN:- 0363-6119
- Animals
- Antibodies -- pharmacology
- Antineoplastic Agents, Phytogenic -- pharmacology
- Cell Count
- Disease Models, Animal
- Disease Progression
- Endotoxemia -- chemically induced
- Fluorescent Antibody Technique
- Heart -- drug effects
- In Vitro Techniques
- Intercellular Adhesion Molecule-1 -- genetics
- Lipopolysaccharides
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myocardium -- metabolism
- Neutrophil Infiltration -- drug effects
- Vascular Cell Adhesion Molecule-1 -- metabolism
- Vinblastine -- pharmacology
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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