Sp1 and TAFII130 transcriptional activity disrupted in early Huntington's disease. [electronic resource]
Producer: 20020723Description: 2238-43 p. digitalISSN:- 1095-9203
- Animals
- Brain -- metabolism
- Caudate Nucleus -- metabolism
- Cell Death
- Cell Line
- Cell Nucleus -- metabolism
- Cells, Cultured
- Corpus Striatum -- cytology
- DNA -- metabolism
- DNA-Binding Proteins -- chemistry
- Down-Regulation
- Gene Expression Regulation
- Humans
- Huntingtin Protein
- Huntington Disease -- genetics
- Mice
- Mice, Transgenic
- Mutation
- Nerve Tissue Proteins -- chemistry
- Neurons -- physiology
- Nuclear Proteins -- chemistry
- Peptides
- Promoter Regions, Genetic
- Rats
- Receptors, Dopamine D2 -- genetics
- Solubility
- Sp1 Transcription Factor -- chemistry
- TATA-Binding Protein Associated Factors
- Transcription Factor TFIID
- Transcription Factors -- chemistry
- Transcription, Genetic
- Transfection
- Trinucleotide Repeat Expansion
- Two-Hybrid System Techniques
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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