Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation. [electronic resource]
Producer: 20020618Description: 799-812 p. digitalISSN:- 0887-6924
- ATP Binding Cassette Transporter, Subfamily B, Member 1 -- metabolism
- Acute Disease
- Bone Marrow Cells -- pathology
- Cell Division
- Daunorubicin
- Drug Resistance, Multiple -- physiology
- Drug Resistance, Neoplasm -- physiology
- HL-60 Cells
- Humans
- JNK Mitogen-Activated Protein Kinases
- Leukemia, Myeloid -- drug therapy
- Mitogen-Activated Protein Kinases -- metabolism
- Phosphorylation
- Proto-Oncogene Proteins c-jun -- metabolism
- Signal Transduction
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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