Growth defect in Grg5 null mice is associated with reduced Ihh signaling in growth plates. [electronic resource]
Producer: 20021010Description: 79-89 p. digitalISSN:- 1058-8388
- Animals
- Bone and Bones -- diagnostic imaging
- Cells, Cultured
- Chondrocytes -- metabolism
- Co-Repressor Proteins
- Drosophila Proteins
- Embryonic Induction
- Female
- Growth Disorders -- metabolism
- Growth Plate -- physiology
- Hedgehog Proteins
- Humans
- Humerus -- cytology
- In Situ Hybridization
- Male
- Membrane Proteins -- genetics
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Osteoblasts -- metabolism
- Patched Receptors
- Proliferating Cell Nuclear Antigen -- metabolism
- Radiography
- Receptors, Cell Surface -- genetics
- Receptors, G-Protein-Coupled
- Repressor Proteins -- genetics
- Signal Transduction -- physiology
- Skull -- cytology
- Smoothened Receptor
- Tibia -- cytology
- Trans-Activators -- genetics
- Transcription Factors
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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