Japanese encephalitis virus infection initiates endoplasmic reticulum stress and an unfolded protein response. [electronic resource]
Producer: 20020510Description: 4162-71 p. digitalISSN:- 0022-538X
- Animals
- Apoptosis
- CCAAT-Enhancer-Binding Proteins -- metabolism
- Cell Line
- Encephalitis Virus, Japanese -- pathogenicity
- Endoplasmic Reticulum -- metabolism
- Gene Expression Regulation
- Mitogen-Activated Protein Kinases -- metabolism
- Molecular Chaperones -- metabolism
- Protein Folding
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Transcription Factor CHOP
- Transcription Factors -- metabolism
- p38 Mitogen-Activated Protein Kinases
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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