Coupling endoplasmic reticulum stress to the cell death program. An Apaf-1-independent intrinsic pathway. [electronic resource]
Producer: 20020725Description: 21836-42 p. digitalISSN:- 0021-9258
- 3T3 Cells
- Animals
- Anticarcinogenic Agents -- pharmacology
- Apoptosis
- Apoptotic Protease-Activating Factor 1
- Blotting, Western
- Brefeldin A -- pharmacology
- Calcium -- metabolism
- Carrier Proteins -- biosynthesis
- Caspase 12
- Caspase 9
- Caspases -- genetics
- Cell Death
- Cell-Free System
- Cells, Cultured
- Endoplasmic Reticulum -- metabolism
- Endoplasmic Reticulum Chaperone BiP
- Enzyme Inhibitors -- pharmacology
- Heat-Shock Proteins
- Mice
- Molecular Chaperones -- biosynthesis
- Mutation
- Plasmids -- metabolism
- Poly Adenosine Diphosphate Ribose -- metabolism
- Precipitin Tests
- Protein Binding
- Protein Biosynthesis
- Protein Structure, Tertiary
- Protein Synthesis Inhibitors -- pharmacology
- Proteins -- metabolism
- Subcellular Fractions
- Tamoxifen -- pharmacology
- Thapsigargin -- pharmacology
- Time Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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