Radiation-induced phosphorylation of Chk1 at S345 is associated with p53-dependent cell cycle arrest pathways. [electronic resource]
Producer: 20020523Description: 171-80 p. digitalISSN:- 1522-8002
- 14-3-3 Proteins
- Alkaloids -- pharmacology
- Antineoplastic Agents -- pharmacology
- Blotting, Western
- Cell Cycle -- drug effects
- Checkpoint Kinase 1
- Colonic Neoplasms -- drug therapy
- Colony-Forming Units Assay
- Cyclin-Dependent Kinase Inhibitor p21
- Cyclins -- metabolism
- DNA -- radiation effects
- DNA Damage
- Drug Resistance
- Enzyme Inhibitors -- pharmacology
- Humans
- Mutation -- genetics
- Phosphorylation
- Protein Kinases -- metabolism
- Signal Transduction
- Staurosporine -- analogs & derivatives
- Tumor Cells, Cultured -- drug effects
- Tumor Suppressor Protein p53 -- metabolism
- Tyrosine 3-Monooxygenase -- metabolism
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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