Chronic exposure of HepG2 cells to excess copper results in depletion of glutathione and induction of metallothionein. [electronic resource]
Producer: 20020626Description: 167-75 p. digitalISSN:- 0887-2333
- Adaptation, Physiological -- drug effects
- Cell Death -- drug effects
- Copper -- toxicity
- Cytosol -- drug effects
- Dose-Response Relationship, Drug
- Enzyme Induction
- Glutathione -- metabolism
- Hepatoblastoma -- enzymology
- Humans
- Liver Neoplasms -- enzymology
- Metallothionein -- biosynthesis
- Oxidative Stress -- drug effects
- Tumor Cells, Cultured
- tert-Butylhydroperoxide -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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