Overgrowth of a mouse model of the Simpson-Golabi-Behmel syndrome is independent of IGF signaling. [electronic resource]
Producer: 20020312Description: 185-206 p. digitalISSN:- 0012-1606
- Abnormalities, Multiple -- genetics
- Animals
- Disease Models, Animal
- Female
- Gene Deletion
- Gene Expression Regulation
- Glypicans
- Heparan Sulfate Proteoglycans -- genetics
- Insulin-Like Growth Factor I -- genetics
- Insulin-Like Growth Factor II -- genetics
- Male
- Mice
- Mice, Knockout
- Receptor, IGF Type 1 -- genetics
- Signal Transduction -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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