In vivo reversal of glutathione deficiency and susceptibility to in vivo dexamethasone-induced apoptosis by N-acetylcysteine and L-2-oxothiazolidine-4-carboxylic acid, but not ascorbic acid, in thymocytes from gamma-glutamyltranspeptidase-deficient knockout mice. [electronic resource]
Producer: 20020225Description: 399-406 p. digitalISSN:- 0003-9861
- Acetylcysteine -- pharmacology
- Animals
- Apoptosis -- drug effects
- Ascorbic Acid -- pharmacology
- Dexamethasone -- pharmacology
- Glutathione -- deficiency
- Mice
- Mice, Knockout
- Pyrrolidonecarboxylic Acid
- Thiazoles -- pharmacology
- Thiazolidines
- Thymus Gland -- cytology
- gamma-Glutamyltransferase -- deficiency
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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