Elevated interleukin-6 during ethanol consumption acts as a potential endogenous protective cytokine against ethanol-induced apoptosis in the liver: involvement of induction of Bcl-2 and Bcl-x(L) proteins. [electronic resource]
Producer: 20020219Description: 32-43 p. digitalISSN:- 0950-9232
- Alcoholism -- metabolism
- Animals
- Apoptosis -- drug effects
- Body Weight -- drug effects
- Cells, Cultured -- drug effects
- Ethanol -- toxicity
- Gene Expression Regulation -- drug effects
- Genes, bcl-2
- Genetic Predisposition to Disease
- Genotype
- Hepatocytes -- drug effects
- Humans
- Interleukin-6 -- blood
- Liver -- drug effects
- Liver Diseases, Alcoholic -- etiology
- Male
- Mice
- Mice, Knockout
- Organ Size -- drug effects
- Proto-Oncogene Proteins c-bcl-2 -- biosynthesis
- Tumor Necrosis Factor-alpha -- toxicity
- bcl-X Protein
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Publication Type: Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S.
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