Increase in de novo HBV DNA integrations in response to oxidative DNA damage or inhibition of poly(ADP-ribosyl)ation. [electronic resource]
Producer: 20020125Description: 217-23 p. digitalISSN:- 0270-9139
- Apoptosis
- Blotting, Southern
- Cloning, Molecular
- DNA Damage
- DNA Repair -- drug effects
- DNA Restriction Enzymes -- metabolism
- DNA, Viral -- genetics
- Deoxyribonucleases, Type II Site-Specific -- metabolism
- Enzyme Inhibitors -- pharmacology
- Hepatitis B virus -- genetics
- Hepatoblastoma
- Humans
- Hydrogen Peroxide -- pharmacology
- In Situ Nick-End Labeling
- Liver Neoplasms
- Oxidative Stress
- Poly Adenosine Diphosphate Ribose -- antagonists & inhibitors
- Tumor Cells, Cultured
- Virus Integration
- Virus Replication
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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