The Chinese hamster FANCG/XRCC9 mutant NM3 fails to express the monoubiquitinated form of the FANCD2 protein, is hypersensitive to a range of DNA damaging agents and exhibits a normal level of spontaneous sister chromatid exchange. [electronic resource]
Producer: 20020204Description: 1939-46 p. digitalISSN:- 0143-3334
- Animals
- Bleomycin -- pharmacology
- CHO Cells
- Cell Line
- Cricetinae
- DNA Damage -- drug effects
- DNA-Binding Proteins -- genetics
- Epoxy Compounds -- pharmacology
- Fanconi Anemia -- genetics
- Fanconi Anemia Complementation Group D2 Protein
- Fanconi Anemia Complementation Group G Protein
- Gamma Rays
- Genetic Complementation Test
- Humans
- Hybrid Cells
- Mechlorethamine -- pharmacology
- Mitomycin -- pharmacology
- Mutagens -- pharmacology
- Mutation -- genetics
- Nuclear Proteins -- chemistry
- Sister Chromatid Exchange -- drug effects
- Ubiquitin -- metabolism
- Ultraviolet Rays
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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