BCR-ABL fails to inhibit apoptosis in U937 myelomonocytic cells expressing a carboxyl-terminal truncated STAT5. [electronic resource]
Producer: 20020116Description: 445-55 p. digitalISSN:- 1042-8194
- Apoptosis -- physiology
- Cell Differentiation
- Cell Division
- Ceramides
- Cloning, Molecular
- DNA-Binding Proteins -- genetics
- Etoposide -- toxicity
- Fusion Proteins, bcr-abl -- genetics
- Genes, abl
- Humans
- Milk Proteins
- Phosphorylation
- Recombinant Proteins -- metabolism
- STAT5 Transcription Factor
- Sequence Deletion
- Trans-Activators -- genetics
- Transfection
- Tumor Necrosis Factor-alpha -- toxicity
- U937 Cells
- fas Receptor -- physiology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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