Inhibition of nitric oxide synthase initiates relapsing remitting experimental autoimmune encephalomyelitis in rats, yet nitric oxide appears to be essential for clinical expression of disease. [electronic resource]
Producer: 20011207Description: 5904-12 p. digitalISSN:- 0022-1767
- Animals
- Arginine -- analogs & derivatives
- Cells, Cultured
- Encephalomyelitis, Autoimmune, Experimental -- enzymology
- Enzyme Inhibitors -- pharmacology
- Kinetics
- Lymph Nodes -- immunology
- Lymphocyte Activation
- Macrophages -- immunology
- Myelin Basic Protein -- immunology
- Nitric Oxide -- physiology
- Nitric Oxide Synthase -- antagonists & inhibitors
- Nitric Oxide Synthase Type II
- Rats
- Rats, Inbred Lew
- Reactive Nitrogen Species -- blood
- Recurrence
- Spinal Cord -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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