APA

McKenna I. M., Ramakrishna G., Diwan B. A., Kang Y., Shiao Y. H., Wakefield L. M., Powell D. A., Anderson L. M. & Jakowlew S. B. (20020116). Heterozygous inactivation of TGF-beta1 increases the susceptibility to chemically induced mouse lung tumorigenesis independently of mutational activation of K-ras. : Toxicology letters.

Chicago

McKenna I M, Ramakrishna G, Diwan B A, Kang Y, Shiao Y H, Wakefield L M, Powell D A, Anderson L M and Jakowlew S B. 20020116. Heterozygous inactivation of TGF-beta1 increases the susceptibility to chemically induced mouse lung tumorigenesis independently of mutational activation of K-ras. : Toxicology letters.

Harvard

McKenna I. M., Ramakrishna G., Diwan B. A., Kang Y., Shiao Y. H., Wakefield L. M., Powell D. A., Anderson L. M. and Jakowlew S. B. (20020116). Heterozygous inactivation of TGF-beta1 increases the susceptibility to chemically induced mouse lung tumorigenesis independently of mutational activation of K-ras. : Toxicology letters.

MLA

McKenna I M, Ramakrishna G, Diwan B A, Kang Y, Shiao Y H, Wakefield L M, Powell D A, Anderson L M and Jakowlew S B. Heterozygous inactivation of TGF-beta1 increases the susceptibility to chemically induced mouse lung tumorigenesis independently of mutational activation of K-ras. : Toxicology letters. 20020116.