Loss of Smad4 function in pancreatic tumors: C-terminal truncation leads to decreased stability. [electronic resource]
Producer: 20011226Description: 43175-81 p. digitalISSN:- 0021-9258
- 3T3 Cells
- Adenocarcinoma -- genetics
- Alleles
- Amino Acids -- chemistry
- Animals
- Blotting, Western
- Cell Line
- Codon
- Codon, Nonsense
- Cycloheximide -- pharmacology
- DNA-Binding Proteins -- chemistry
- Genes, Dominant
- Humans
- Loss of Heterozygosity
- Mesoderm -- metabolism
- Mice
- Models, Molecular
- Mutation
- Nerve Growth Factors
- Pancreatic Neoplasms -- chemistry
- Plasmids -- metabolism
- Precipitin Tests
- Protein Binding
- Protein Structure, Tertiary
- Protein Synthesis Inhibitors -- pharmacology
- RNA, Messenger -- metabolism
- Ribonucleases -- metabolism
- Signal Transduction
- Smad Proteins
- Smad2 Protein
- Smad4 Protein
- Time Factors
- Trans-Activators -- chemistry
- Transcription, Genetic
- Transforming Growth Factor beta -- metabolism
- Ubiquitin -- pharmacology
- Xenopus
- Xenopus Proteins
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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