Blocking Smad7 restores TGF-beta1 signaling in chronic inflammatory bowel disease. [electronic resource]
Producer: 20010920Description: 601-9 p. digitalISSN:- 0021-9738
- Activin Receptors, Type I
- Adolescent
- Adult
- Cells, Cultured -- drug effects
- Child
- Colitis, Ulcerative -- metabolism
- Crohn Disease -- metabolism
- Cytokines -- biosynthesis
- DNA-Binding Proteins -- antagonists & inhibitors
- Female
- Gene Expression Regulation -- drug effects
- Humans
- Inflammatory Bowel Diseases -- metabolism
- Interferon-gamma -- biosynthesis
- Intestinal Mucosa -- drug effects
- Male
- Middle Aged
- Oligodeoxyribonucleotides, Antisense -- pharmacology
- Organ Culture Techniques
- Phosphorylation
- Protein Processing, Post-Translational
- Protein Serine-Threonine Kinases -- drug effects
- Receptor, Transforming Growth Factor-beta Type I
- Receptors, Transforming Growth Factor beta -- drug effects
- Signal Transduction -- drug effects
- Smad3 Protein
- Smad7 Protein
- Trans-Activators -- antagonists & inhibitors
- Transforming Growth Factor beta -- physiology
- Transforming Growth Factor beta1
- Tumor Necrosis Factor-alpha -- biosynthesis
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.