Tumor necrosis factor receptor deletion reduces nuclear factor-kappaB activation, cellular inhibitor of apoptosis protein 2 expression, and functional recovery after traumatic spinal cord injury. [electronic resource]
Producer: 20010920Description: 6617-25 p. digitalISSN:- 1529-2401
- Animals
- Antigens, CD -- genetics
- Apoptosis
- Axons -- pathology
- Baculoviral IAP Repeat-Containing 3 Protein
- Caspase 3
- Caspases -- metabolism
- Female
- In Situ Nick-End Labeling
- Inhibitor of Apoptosis Proteins
- Magnetic Resonance Imaging
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Mice, Transgenic
- Motor Activity
- Myelin Sheath -- metabolism
- NF-kappa B -- metabolism
- Proteins -- metabolism
- Receptors, Tumor Necrosis Factor -- deficiency
- Receptors, Tumor Necrosis Factor, Type I
- Receptors, Tumor Necrosis Factor, Type II
- Recovery of Function
- Signal Transduction -- physiology
- Spinal Cord -- metabolism
- Spinal Cord Injuries -- genetics
- Ubiquitin-Protein Ligases
- Wounds, Nonpenetrating
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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