Oxidative stress-mediated cardiac cell death is a major determinant of ventricular dysfunction and failure in dog dilated cardiomyopathy. [electronic resource]
Producer: 20010823Description: 279-86 p. digitalISSN:- 1524-4571
- Adaptor Proteins, Signal Transducing
- Adaptor Proteins, Vesicular Transport
- Animals
- Apoptosis
- Blotting, Western
- Cardiac Pacing, Artificial
- Cardiomyopathy, Dilated -- pathology
- Caspase 3
- Caspase 9
- Caspases -- metabolism
- Cytochrome c Group -- metabolism
- Disease Models, Animal
- Dogs
- Enzyme Activation -- physiology
- Hemodynamics
- Immunohistochemistry
- In Situ Nick-End Labeling
- Myocardium -- metabolism
- Oxidative Stress
- Protein Biosynthesis
- Reactive Oxygen Species -- metabolism
- Shc Signaling Adaptor Proteins
- Tumor Suppressor Protein p53 -- metabolism
- Tyrosine -- analogs & derivatives
- Ventricular Dysfunction -- etiology
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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