Activation of NF-kappa B by nontypeable Hemophilus influenzae is mediated by toll-like receptor 2-TAK1-dependent NIK-IKK alpha /beta-I kappa B alpha and MKK3/6-p38 MAP kinase signaling pathways in epithelial cells. [electronic resource]
Producer: 20010830Description: 8774-9 p. digitalISSN:- 0027-8424
- Calcium-Calmodulin-Dependent Protein Kinases -- metabolism
- DNA-Binding Proteins -- metabolism
- Drosophila Proteins
- Epithelial Cells
- Haemophilus influenzae -- physiology
- HeLa Cells
- Humans
- I-kappa B Kinase
- I-kappa B Proteins
- MAP Kinase Kinase 3
- MAP Kinase Kinase 6
- MAP Kinase Kinase Kinases -- metabolism
- MAP Kinase Signaling System
- Membrane Glycoproteins -- metabolism
- Mitogen-Activated Protein Kinase Kinases -- metabolism
- Mitogen-Activated Protein Kinases -- metabolism
- NF-KappaB Inhibitor alpha
- NF-kappa B -- metabolism
- Phosphorylation
- Protein Serine-Threonine Kinases -- metabolism
- Protein-Tyrosine Kinases -- metabolism
- Receptors, Cell Surface -- metabolism
- Toll-Like Receptor 2
- Toll-Like Receptors
- Tumor Cells, Cultured
- p38 Mitogen-Activated Protein Kinases
- NF-kappaB-Inducing Kinase
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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