Age-related impairment of synaptic transmission but normal long-term potentiation in transgenic mice that overexpress the human APP695SWE mutant form of amyloid precursor protein. [electronic resource]
Producer: 20010726Description: 4691-8 p. digitalISSN:- 1529-2401
- Aging -- genetics
- Alzheimer Disease -- genetics
- Amyloid beta-Protein Precursor -- biosynthesis
- Animals
- Disease Models, Animal
- Electric Stimulation
- Excitatory Amino Acid Antagonists -- pharmacology
- Excitatory Postsynaptic Potentials -- drug effects
- Genetic Predisposition to Disease
- Hippocampus -- drug effects
- Humans
- In Vitro Techniques
- Kynurenic Acid -- pharmacology
- Long-Term Potentiation -- genetics
- Mice
- Mice, Transgenic
- Mutation
- Neuronal Plasticity
- Plaque, Amyloid -- pathology
- Synaptic Transmission -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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