Activation of NF-kappa B via the Ikappa B kinase complex is both essential and sufficient for proinflammatory gene expression in primary endothelial cells. [electronic resource]
Producer: 20010823Description: 28451-8 p. digitalISSN:- 0021-9258
- Blotting, Western
- Cells, Cultured
- Chemokine CCL2 -- metabolism
- E-Selectin -- metabolism
- Endothelium, Vascular -- cytology
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- Flow Cytometry
- Genes, Dominant
- Humans
- I-kappa B Kinase
- Inflammation -- metabolism
- Intercellular Adhesion Molecule-1 -- metabolism
- Interferon-gamma -- metabolism
- Interleukin-8 -- metabolism
- Mutation
- NF-kappa B -- genetics
- Phosphorylation
- Protein Serine-Threonine Kinases -- genetics
- Retroviridae -- genetics
- Signal Transduction
- Tumor Necrosis Factor-alpha -- metabolism
- Umbilical Veins -- cytology
- Up-Regulation
- Vascular Cell Adhesion Molecule-1 -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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