Tyrosine kinase-dependent, phosphatidylinositol 3'-kinase, and mitogen-activated protein kinase-independent signaling pathways prevent lung adenocarcinoma cells from anoikis. [electronic resource]
Producer: 20010426Description: 2439-44 p. digitalISSN:- 0008-5472
- Adenocarcinoma -- enzymology
- Anoikis -- physiology
- Cell Adhesion -- physiology
- Cell Survival -- physiology
- Enzyme Inhibitors -- pharmacology
- Genistein -- pharmacology
- Humans
- Lung Neoplasms -- enzymology
- MAP Kinase Signaling System -- physiology
- Mitogen-Activated Protein Kinases -- antagonists & inhibitors
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoinositide-3 Kinase Inhibitors
- Phosphorylation
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- antagonists & inhibitors
- Proto-Oncogene Proteins c-akt
- Substrate Specificity
- Tumor Cells, Cultured
- Tyrosine -- metabolism
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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