Activation of protein kinase A and atypical protein kinase C by A(2A) adenosine receptors antagonizes apoptosis due to serum deprivation in PC12 cells. [electronic resource]
Producer: 20010614Description: 13838-46 p. digitalISSN:- 0021-9258
- Adenosine -- analogs & derivatives
- Animals
- Apoptosis
- Blotting, Western
- Cell Membrane -- metabolism
- Cell Nucleus -- metabolism
- Cell Survival
- Chromones -- pharmacology
- Colforsin -- pharmacology
- Coloring Agents -- pharmacology
- Culture Media, Serum-Free -- metabolism
- Cyclic AMP-Dependent Protein Kinases -- metabolism
- Cytosol -- metabolism
- DNA Fragmentation
- Dose-Response Relationship, Drug
- Down-Regulation
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- Flow Cytometry
- Genes, Dominant
- MAP Kinase Signaling System
- Models, Biological
- Morpholines -- pharmacology
- PC12 Cells
- Phenethylamines -- pharmacology
- Phosphoinositide-3 Kinase Inhibitors
- Phosphoprotein Phosphatases -- metabolism
- Protein Kinase C -- metabolism
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins c-akt
- Rats
- Receptor, Adenosine A2A
- Receptors, Purinergic P1 -- metabolism
- Signal Transduction
- Tetrazolium Salts -- pharmacology
- Thiazoles -- pharmacology
- Time Factors
- Transfection
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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