Targeting Pyk2 to beta 1-integrin-containing focal contacts rescues fibronectin-stimulated signaling and haptotactic motility defects of focal adhesion kinase-null cells. [electronic resource]
Producer: 20010301Description: 97-110 p. digitalISSN:- 0021-9525
- Animals
- Cell Movement -- physiology
- Cells, Cultured
- Cytoskeletal Proteins -- metabolism
- Enzyme Activation
- Fibroblasts -- cytology
- Fibronectins -- metabolism
- Focal Adhesion Kinase 1
- Focal Adhesion Kinase 2
- Focal Adhesion Protein-Tyrosine Kinases
- Gene Expression
- Humans
- Integrin beta1 -- metabolism
- JNK Mitogen-Activated Protein Kinases
- Mice
- Mice, Knockout
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinases -- metabolism
- Paxillin
- Phosphoproteins -- metabolism
- Protein-Tyrosine Kinases -- genetics
- Recombinant Fusion Proteins -- genetics
- Signal Transduction
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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