Characterization of the increase in [Ca(2+)](i) during hypotonic shock and the involvement of Ca(2+)-activated K(+) channels in the regulatory volume decrease in human osteoblast-like cells. [electronic resource]
Producer: 20010125Description: 11-20 p. digitalISSN:- 0022-2631
- Calcium -- metabolism
- Cell Line
- Charybdotoxin -- pharmacology
- Electrophysiology
- Humans
- Hypotonic Solutions
- Indoles -- pharmacology
- Intermediate-Conductance Calcium-Activated Potassium Channels
- Large-Conductance Calcium-Activated Potassium Channel alpha Subunits
- Large-Conductance Calcium-Activated Potassium Channels
- Osteoblasts -- drug effects
- Potassium Channel Blockers
- Potassium Channels -- physiology
- Potassium Channels, Calcium-Activated
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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