Ir gene defects may reflect a regulatory imbalance. I. Helper T cell activity revealed in a strain whose lack of response is controlled by suppression. [electronic resource]

By:

Araneo, B A

Contributor(s):

Yowell, R L
Sercarz, E E

Producer: 19791026Description: 961-7 p. digitalISSN:

0022-1767

Subject(s):

Animals
Birds
Chickens
Dose-Response Relationship, Immunologic
Female
Genes, MHC Class II
Immunosuppression Therapy
Kinetics
Lymph Nodes -- immunology
Mice
Mice, Inbred C57BL
Mice, Inbred CBA
Mice, Inbred DBA
Muramidase -- immunology
Perissodactyla
Phenotype
T-Lymphocytes -- immunology

In: Journal of immunology (Baltimore, Md. : 1950) vol. 123

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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.

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Ir gene defects may reflect a regulatory imbalance. I. Helper T cell activity revealed in a strain whose lack of response is controlled by suppression.

APA

Araneo B. A., Yowell R. L. & Sercarz E. E. (19791026). Ir gene defects may reflect a regulatory imbalance. I. Helper T cell activity revealed in a strain whose lack of response is controlled by suppression. : Journal of immunology (Baltimore, Md. : 1950).

Chicago

Araneo B A, Yowell R L and Sercarz E E. 19791026. Ir gene defects may reflect a regulatory imbalance. I. Helper T cell activity revealed in a strain whose lack of response is controlled by suppression. : Journal of immunology (Baltimore, Md. : 1950).

Harvard

Araneo B. A., Yowell R. L. and Sercarz E. E. (19791026). Ir gene defects may reflect a regulatory imbalance. I. Helper T cell activity revealed in a strain whose lack of response is controlled by suppression. : Journal of immunology (Baltimore, Md. : 1950).

MLA

Araneo B A, Yowell R L and Sercarz E E. Ir gene defects may reflect a regulatory imbalance. I. Helper T cell activity revealed in a strain whose lack of response is controlled by suppression. : Journal of immunology (Baltimore, Md. : 1950). 19791026.

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