Kupffer cells mediate increased anoxic hepatocellular killing from hyperosmolarity by an oxygen- and prostaglandin-independent mechanism. [electronic resource]
Producer: 20001102Description: 95-100 p. digitalISSN:- 0378-4274
- Animals
- Eating
- Fasting
- Gadolinium -- pharmacology
- Glycolysis -- drug effects
- Hepatocytes -- cytology
- Hypoxia -- physiopathology
- In Vitro Techniques
- Indomethacin -- pharmacology
- Kupffer Cells -- cytology
- L-Lactate Dehydrogenase -- drug effects
- Liver -- cytology
- Male
- Oxygen -- physiology
- Oxygen Consumption -- drug effects
- Prostaglandins -- biosynthesis
- Rats
- Rats, Sprague-Dawley
- Saline Solution, Hypertonic -- pharmacology
- Time Factors
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Publication Type: Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S.
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