Deficiency of complement defense protein CD59 may contribute to neurodegeneration in Alzheimer's disease. [electronic resource]
Producer: 20010607Description: 7505-9 p. digitalISSN:- 1529-2401
- Aged
- Aged, 80 and over
- Alzheimer Disease -- immunology
- Amyloid beta-Peptides -- pharmacology
- CD59 Antigens -- genetics
- Cell Differentiation -- drug effects
- Cell Survival -- drug effects
- Cells, Cultured
- Complement C9 -- genetics
- Complement System Proteins -- pharmacology
- Dose-Response Relationship, Drug
- Female
- Frontal Lobe -- metabolism
- Gene Expression -- drug effects
- Glycosylphosphatidylinositols -- metabolism
- Hippocampus -- metabolism
- Humans
- Male
- Neurons -- drug effects
- Peptide Fragments -- pharmacology
- Phosphatidylinositol Diacylglycerol-Lyase
- Phosphopyruvate Hydratase -- metabolism
- RNA, Messenger -- metabolism
- Ribonucleases -- deficiency
- Synaptophysin -- metabolism
- Transfection
- Type C Phospholipases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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