Association of B7-1 co-stimulation with the development of graft arterial disease. Studies using mice lacking B7-1, B7-2, or B7-1/B7-2. [electronic resource]
Producer: 20000823Description: 473-84 p. digitalISSN:- 0002-9440
- Animals
- Antigen-Presenting Cells -- physiology
- Antigens, CD -- analysis
- B-Lymphocytes -- chemistry
- B7-1 Antigen -- analysis
- B7-2 Antigen
- CD4-Positive T-Lymphocytes -- chemistry
- CD8-Positive T-Lymphocytes -- chemistry
- Chemokines -- genetics
- Cytokines -- genetics
- Female
- Flow Cytometry
- Gene Expression Regulation
- Genotype
- Graft Occlusion, Vascular -- etiology
- Graft Rejection -- complications
- Heart Transplantation
- Histocompatibility Antigens Class II -- analysis
- Immunohistochemistry
- Macrophages -- chemistry
- Male
- Membrane Glycoproteins -- analysis
- Mice
- Mice, Inbred C57BL
- Time Factors
- Transplantation, Homologous
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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