Phosphorylation of elk-1 by MEK/ERK pathway is necessary for c-fos gene activation during cardiac myocyte hypertrophy. [electronic resource]
Producer: 20001006Description: 1447-57 p. digitalISSN:- 0022-2828
- Adrenergic alpha-Agonists -- pharmacology
- Animals
- Animals, Newborn
- Blotting, Western
- Cardiomegaly -- pathology
- Cell Nucleus -- metabolism
- Cells, Cultured
- Cytosol -- metabolism
- DNA-Binding Proteins -- metabolism
- Dose-Response Relationship, Drug
- Electrophoresis, Polyacrylamide Gel
- Enzyme Inhibitors -- pharmacology
- Flavonoids -- pharmacology
- Genes, Reporter
- Imidazoles -- pharmacology
- MAP Kinase Kinase 1
- Microscopy, Confocal
- Mitogen-Activated Protein Kinase Kinases -- metabolism
- Mitogen-Activated Protein Kinases -- metabolism
- Myocardium -- cytology
- Nuclear Proteins -- metabolism
- Phenylephrine -- pharmacology
- Phosphorylation
- Plasmids -- metabolism
- Promoter Regions, Genetic
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-fos -- genetics
- Pyridines -- pharmacology
- Rats
- Rats, Sprague-Dawley
- Serum Response Factor
- Time Factors
- Transcription Factors
- Transfection
- ets-Domain Protein Elk-1
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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