APA

Redwood C., Lohmann K., Bing W., Esposito G. M., Elliott K., Abdulrazzak H., Knott A., Purcell I., Marston S. & Watkins H. (20000714). Investigation of a truncated cardiac troponin T that causes familial hypertrophic cardiomyopathy: Ca(2+) regulatory properties of reconstituted thin filaments depend on the ratio of mutant to wild-type protein. : Circulation research.

Chicago

Redwood C, Lohmann K, Bing W, Esposito G M, Elliott K, Abdulrazzak H, Knott A, Purcell I, Marston S and Watkins H. 20000714. Investigation of a truncated cardiac troponin T that causes familial hypertrophic cardiomyopathy: Ca(2+) regulatory properties of reconstituted thin filaments depend on the ratio of mutant to wild-type protein. : Circulation research.

Harvard

Redwood C., Lohmann K., Bing W., Esposito G. M., Elliott K., Abdulrazzak H., Knott A., Purcell I., Marston S. and Watkins H. (20000714). Investigation of a truncated cardiac troponin T that causes familial hypertrophic cardiomyopathy: Ca(2+) regulatory properties of reconstituted thin filaments depend on the ratio of mutant to wild-type protein. : Circulation research.

MLA

Redwood C, Lohmann K, Bing W, Esposito G M, Elliott K, Abdulrazzak H, Knott A, Purcell I, Marston S and Watkins H. Investigation of a truncated cardiac troponin T that causes familial hypertrophic cardiomyopathy: Ca(2+) regulatory properties of reconstituted thin filaments depend on the ratio of mutant to wild-type protein. : Circulation research. 20000714.