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  2. Details for: Rapid destruction of human Cdc25A in response to DNA damage.
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Rapid destruction of human Cdc25A in response to DNA damage. [electronic resource]

By:
  • Mailand, N
Contributor(s):
  • Falck, J
  • Lukas, C
  • Syljuâsen, R G
  • Welcker, M
  • Bartek, J
  • Lukas, J
Producer: 20000608Description: 1425-9 p. digitalISSN:
  • 0036-8075
Subject(s):
  • CDC2-CDC28 Kinases
  • Cell Line
  • Cell Survival
  • Checkpoint Kinase 1
  • Cyclin E -- metabolism
  • Cyclin-Dependent Kinase 2
  • Cyclin-Dependent Kinases -- antagonists & inhibitors
  • Cysteine Endopeptidases -- metabolism
  • DNA Damage
  • DNA Repair
  • DNA Replication
  • G1 Phase
  • Humans
  • Multienzyme Complexes -- metabolism
  • Phosphorylation
  • Phosphotyrosine -- metabolism
  • Proteasome Endopeptidase Complex
  • Protein Kinase Inhibitors
  • Protein Kinases -- metabolism
  • Protein Serine-Threonine Kinases -- antagonists & inhibitors
  • Recombinant Fusion Proteins -- metabolism
  • S Phase
  • Transfection
  • Tumor Cells, Cultured
  • Tumor Suppressor Protein p53 -- metabolism
  • Ultraviolet Rays
  • cdc25 Phosphatases -- genetics
Online resources:
  • Available from publisher's website
In: Science (New York, N.Y.) vol. 288
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't

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Rapid destruction of human Cdc25A in response to DNA damage.

APA

Mailand N., Falck J., Lukas C., Syljuâsen R. G., Welcker M., Bartek J. & Lukas J. (20000608). Rapid destruction of human Cdc25A in response to DNA damage. : Science (New York, N.Y.).

Chicago

Mailand N, Falck J, Lukas C, Syljuâsen R G, Welcker M, Bartek J and Lukas J. 20000608. Rapid destruction of human Cdc25A in response to DNA damage. : Science (New York, N.Y.).

Harvard

Mailand N., Falck J., Lukas C., Syljuâsen R. G., Welcker M., Bartek J. and Lukas J. (20000608). Rapid destruction of human Cdc25A in response to DNA damage. : Science (New York, N.Y.).

MLA

Mailand N, Falck J, Lukas C, Syljuâsen R G, Welcker M, Bartek J and Lukas J. Rapid destruction of human Cdc25A in response to DNA damage. : Science (New York, N.Y.). 20000608.

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