E2F1 mediates death of B-amyloid-treated cortical neurons in a manner independent of p53 and dependent on Bax and caspase 3. [electronic resource]
Producer: 20000510Description: 11553-60 p. digitalISSN:- 0021-9258
- Amyloid beta-Peptides -- pharmacology
- Animals
- Apoptosis
- Carrier Proteins
- Caspase 3
- Caspases -- metabolism
- Cell Cycle Proteins -- physiology
- Cells, Cultured
- Cerebral Cortex -- cytology
- Coumarins -- pharmacology
- DNA-Binding Proteins -- physiology
- E2F Transcription Factors
- E2F1 Transcription Factor
- Enzyme Activation
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Neurons -- drug effects
- Oligopeptides -- pharmacology
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-bcl-2
- Retinoblastoma-Binding Protein 1
- Transcription Factor DP1
- Transcription Factors -- physiology
- Tumor Suppressor Protein p53 -- physiology
- bcl-2-Associated X Protein
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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