Modulation of hypoxia-inducible factor 1alpha expression by the epidermal growth factor/phosphatidylinositol 3-kinase/PTEN/AKT/FRAP pathway in human prostate cancer cells: implications for tumor angiogenesis and therapeutics. [electronic resource]
Producer: 20000504Description: 1541-5 p. digitalISSN:- 0008-5472
- Carrier Proteins
- Chromones -- pharmacology
- Culture Media, Serum-Free -- pharmacology
- DNA-Binding Proteins -- biosynthesis
- Endothelial Growth Factors -- biosynthesis
- Enzyme Inhibitors -- pharmacology
- Enzyme-Linked Immunosorbent Assay
- Epidermal Growth Factor -- genetics
- Humans
- Hypoxia -- physiopathology
- Hypoxia-Inducible Factor 1
- Hypoxia-Inducible Factor 1, alpha Subunit
- Immunophilins -- genetics
- Lymphokines -- biosynthesis
- Male
- Morpholines -- pharmacology
- Neovascularization, Pathologic -- metabolism
- Nuclear Proteins -- biosynthesis
- PTEN Phosphohydrolase
- Phosphatidylinositol 3-Kinases -- genetics
- Phosphoinositide-3 Kinase Inhibitors
- Phosphoric Monoester Hydrolases -- genetics
- Phosphotransferases (Alcohol Group Acceptor)
- Prostatic Neoplasms -- drug therapy
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins c-akt
- Signal Transduction -- drug effects
- TOR Serine-Threonine Kinases
- Tetradecanoylphorbol Acetate -- pharmacology
- Transcription Factors
- Tumor Cells, Cultured -- drug effects
- Tumor Suppressor Proteins
- Vascular Endothelial Growth Factor A
- Vascular Endothelial Growth Factors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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