APA

Pece-Barbara N., Cymerman U., Vera S., Marchuk D. A. & Letarte M. (19991214). Expression analysis of four endoglin missense mutations suggests that haploinsufficiency is the predominant mechanism for hereditary hemorrhagic telangiectasia type 1. : Human molecular genetics.

Chicago

Pece-Barbara N, Cymerman U, Vera S, Marchuk D A and Letarte M. 19991214. Expression analysis of four endoglin missense mutations suggests that haploinsufficiency is the predominant mechanism for hereditary hemorrhagic telangiectasia type 1. : Human molecular genetics.

Harvard

Pece-Barbara N., Cymerman U., Vera S., Marchuk D. A. and Letarte M. (19991214). Expression analysis of four endoglin missense mutations suggests that haploinsufficiency is the predominant mechanism for hereditary hemorrhagic telangiectasia type 1. : Human molecular genetics.

MLA

Pece-Barbara N, Cymerman U, Vera S, Marchuk D A and Letarte M. Expression analysis of four endoglin missense mutations suggests that haploinsufficiency is the predominant mechanism for hereditary hemorrhagic telangiectasia type 1. : Human molecular genetics. 19991214.