APA

Obici L., Bellotti V., Mangione P., Stoppini M., Arbustini E., Verga L., Zorzoli I., Anesi E., Zanotti G., Campana C., Viganò M. & Merlini G. (19991007). The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide. : The American journal of pathology.

Chicago

Obici L, Bellotti V, Mangione P, Stoppini M, Arbustini E, Verga L, Zorzoli I, Anesi E, Zanotti G, Campana C, Viganò M and Merlini G. 19991007. The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide. : The American journal of pathology.

Harvard

Obici L., Bellotti V., Mangione P., Stoppini M., Arbustini E., Verga L., Zorzoli I., Anesi E., Zanotti G., Campana C., Viganò M. and Merlini G. (19991007). The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide. : The American journal of pathology.

MLA

Obici L, Bellotti V, Mangione P, Stoppini M, Arbustini E, Verga L, Zorzoli I, Anesi E, Zanotti G, Campana C, Viganò M and Merlini G. The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide. : The American journal of pathology. 19991007.