Norepinephrine-mediated inhibition of antitumor cytotoxic T lymphocyte generation involves a beta-adrenergic receptor mechanism and decreased TNF-alpha gene expression. [electronic resource]
Producer: 19990914Description: 2492-9 p. digitalISSN:- 0022-1767
- Animals
- Antigens, Differentiation -- biosynthesis
- CD3 Complex -- biosynthesis
- CD4-Positive T-Lymphocytes -- metabolism
- CD8-Positive T-Lymphocytes -- metabolism
- Cytotoxicity, Immunologic -- genetics
- Female
- Gene Expression Regulation, Neoplastic -- drug effects
- Intracellular Fluid -- drug effects
- Lymphocyte Activation -- genetics
- Lymphocyte Subsets -- metabolism
- Macrophages -- drug effects
- Melphalan -- toxicity
- Mice
- Mice, Inbred BALB C
- Neoplasm Transplantation
- Norepinephrine -- pharmacology
- Plasmacytoma -- chemically induced
- Propranolol -- pharmacology
- RNA, Messenger -- biosynthesis
- Receptors, Adrenergic, beta -- physiology
- Spleen -- immunology
- T-Lymphocytes, Cytotoxic -- immunology
- Tumor Cells, Cultured
- Tumor Necrosis Factor-alpha -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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