TrkB mutant lacking the amino-terminal half of the extracellular portion acts as a functional brain-derived neurotrophic factor receptor. [electronic resource]
Producer: 19990928Description: 104-10 p. digitalISSN:- 0006-3002
- 3T3 Cells
- Animals
- Binding Sites -- genetics
- Brain-Derived Neurotrophic Factor -- metabolism
- COS Cells
- In Vitro Techniques
- Mice
- Peptide Fragments -- chemistry
- Protein Conformation
- Receptor Protein-Tyrosine Kinases -- chemistry
- Receptor, Ciliary Neurotrophic Factor
- Receptors, Nerve Growth Factor -- chemistry
- Recombinant Proteins -- chemistry
- Sequence Deletion
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Publication Type: Comparative Study; Journal Article
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