Growth inhibition by CDK-cyclin and PCNA binding domains of p21 occurs by distinct mechanisms and is regulated by ubiquitin-proteasome pathway. [electronic resource]
Producer: 19990701Description: 3290-302 p. digitalISSN:- 0950-9232
- 3T3 Cells
- Animals
- Binding Sites
- CDC2-CDC28 Kinases
- Carrier Proteins
- Cell Cycle Proteins
- Cell Division
- Cyclin-Dependent Kinase 2
- Cyclin-Dependent Kinase Inhibitor p21
- Cyclin-Dependent Kinases -- metabolism
- Cyclins -- genetics
- Cysteine Endopeptidases -- metabolism
- DNA-Binding Proteins
- E2F Transcription Factors
- Genetic Vectors
- Humans
- Mice
- Multienzyme Complexes -- metabolism
- Mutagenesis
- Proliferating Cell Nuclear Antigen -- metabolism
- Proteasome Endopeptidase Complex
- Protein Serine-Threonine Kinases -- metabolism
- Recombinant Fusion Proteins -- genetics
- Retinoblastoma-Binding Protein 1
- S Phase
- Transcription Factor DP1
- Transcription Factors -- metabolism
- Transcription, Genetic
- Tumor Cells, Cultured
- Ubiquitins -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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