Complement-mediated injury reversibly disrupts glomerular epithelial cell actin microfilaments and focal adhesions. [electronic resource]
Producer: 19990615Description: 1763-75 p. digitalISSN:- 0085-2538
- Actin Cytoskeleton -- chemistry
- Actins -- metabolism
- Animals
- Blotting, Western
- Cell Adhesion -- immunology
- Cell Adhesion Molecules -- analysis
- Cell Line
- Complement Membrane Attack Complex -- chemistry
- Complement System Proteins -- immunology
- Cycloheximide -- pharmacology
- Cytoskeletal Proteins -- analysis
- Epithelial Cells -- chemistry
- Fluorescent Antibody Technique
- Focal Adhesion Kinase 1
- Focal Adhesion Protein-Tyrosine Kinases
- Glomerulonephritis -- immunology
- Immunoglobulin G -- pharmacology
- Integrin alpha3beta1
- Integrins -- analysis
- Kidney Glomerulus -- cytology
- Paxillin
- Phosphoproteins -- analysis
- Phosphotyrosine -- analysis
- Protein Synthesis Inhibitors -- pharmacology
- Protein-Tyrosine Kinases -- analysis
- Rats
- Receptors, Laminin -- analysis
- Talin -- analysis
- Vanadates -- pharmacology
- Vinculin -- analysis
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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