Activation of G0/G1 switch gene 2 by endoplasmic reticulum stress enhances hepatic steatosis. (Record no. 29867968)
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| 000 -LEADER | |
|---|---|
| fixed length control field | 01475 a2200421 4500 |
| 005 - DATE AND TIME OF LATEST TRANSACTION | |
| control field | 20250518050044.0 |
| 008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION | |
| fixed length control field | 202002s 0 0 eng d |
| 022 ## - INTERNATIONAL STANDARD SERIAL NUMBER | |
| International Standard Serial Number | 1532-8600 |
| 024 7# - OTHER STANDARD IDENTIFIER | |
| Standard number or code | 10.1016/j.metabol.2019.06.015 |
| Source of number or code | doi |
| 040 ## - CATALOGING SOURCE | |
| Original cataloging agency | NLM |
| Language of cataloging | eng |
| Transcribing agency | NLM |
| 100 1# - MAIN ENTRY--PERSONAL NAME | |
| Personal name | Ma, Yunqin |
| 264 #0 - PRODUCTION, PUBLICATION, DISTRIBUTION, MANUFACTURE, AND COPYRIGHT NOTICE | |
| Date of production, publication, distribution, manufacture, or copyright notice | 20200219 |
| 245 00 - TITLE STATEMENT | |
| Title | Activation of G0/G1 switch gene 2 by endoplasmic reticulum stress enhances hepatic steatosis. |
| Medium | [electronic resource] |
| 260 ## - PUBLICATION, DISTRIBUTION, ETC. | |
| Name of publisher, distributor, etc. | Metabolism: clinical and experimental |
| Date of publication, distribution, etc. | 10 2019 |
| 300 ## - PHYSICAL DESCRIPTION | |
| Extent | 32-44 p. |
| Other physical details | digital |
| 500 ## - GENERAL NOTE | |
| General note | Publication Type: Journal Article; Research Support, Non-U.S. Gov't |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | 5' Untranslated Regions |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Activating Transcription Factor 4 |
| General subdivision | metabolism |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Animals |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Cell Cycle Proteins |
| General subdivision | genetics |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Endoplasmic Reticulum Stress |
| General subdivision | genetics |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Hep G2 Cells |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Humans |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Mice |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Mice, Inbred C57BL |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Non-alcoholic Fatty Liver Disease |
| General subdivision | etiology |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Transcriptional Activation |
| 650 04 - SUBJECT ADDED ENTRY--TOPICAL TERM | |
| Topical term or geographic name entry element | Unfolded Protein Response |
| 700 1# - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Zhang, Mingliang |
| 700 1# - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Yu, Haoyong |
| 700 1# - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Lu, Junxi |
| 700 1# - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Cheng, Kenneth K Y |
| 700 1# - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Zhou, Jian |
| 700 1# - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Chen, Haibing |
| 700 1# - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Jia, Weiping |
| 773 0# - HOST ITEM ENTRY | |
| Title | Metabolism: clinical and experimental |
| Related parts | vol. 99 |
| -- | p. 32-44 |
| 856 40 - ELECTRONIC LOCATION AND ACCESS | |
| Uniform Resource Identifier | <a href="https://doi.org/10.1016/j.metabol.2019.06.015">https://doi.org/10.1016/j.metabol.2019.06.015</a> |
| Public note | Available from publisher's website |
No items available.