Macrophage inflammatory protein-1 alpha production in lipopolysaccharide-stimulated human adherent blood mononuclear cells is inhibited by the nitric oxide synthase inhibitor N(G)-monomethyl-L-arginine.

Mühl, H

Macrophage inflammatory protein-1 alpha production in lipopolysaccharide-stimulated human adherent blood mononuclear cells is inhibited by the nitric oxide synthase inhibitor N(G)-monomethyl-L-arginine. [electronic resource] - Journal of immunology (Baltimore, Md. : 1950) Nov 1997 - 5063-9 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

0022-1767


Animals
Arginine--metabolism
Cell Adhesion--immunology
Cell Line
Chemokine CCL2--biosynthesis
Chemokine CCL4
Chemokine CCL5--biosynthesis
Gene Expression Regulation--drug effects
Humans
Interleukin-1--metabolism
Interleukin-10--metabolism
Interleukin-8--biosynthesis
Leukocytes, Mononuclear--enzymology
Lipopolysaccharides--pharmacology
Macrophage Inflammatory Proteins--biosynthesis
Mice
Nitric Oxide--physiology
Nitric Oxide Synthase--antagonists & inhibitors
Substrate Specificity
Tumor Necrosis Factor-alpha--metabolism
omega-N-Methylarginine--pharmacology