The kidneys of mice with autoimmune disease acquire a hypofibrinolytic/procoagulant state that correlates with the development of glomerulonephritis and tissue microthrombosis.

Yamamoto, K

The kidneys of mice with autoimmune disease acquire a hypofibrinolytic/procoagulant state that correlates with the development of glomerulonephritis and tissue microthrombosis. [electronic resource] - The American journal of pathology Sep 1997 - 725-34 p. digital

Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.

0002-9440


Aging
Animals
Brain--metabolism
Female
Fibrin--metabolism
In Situ Hybridization
Kidney--drug effects
Lipopolysaccharides--pharmacology
Liver--metabolism
Lung--metabolism
Lupus Vulgaris--metabolism
Mice
Mice, Inbred Strains
Myocardium--metabolism
Plasminogen Activator Inhibitor 1--blood
Polymerase Chain Reaction
RNA, Messenger--metabolism
Spleen--metabolism
Thromboplastin--genetics
Tissue Distribution
Tissue Plasminogen Activator--genetics
Urokinase-Type Plasminogen Activator--genetics