Biochemical and genetic data suggest that InhA is not the primary target for activated isoniazid in Mycobacterium tuberculosis.

Mdluli, K

Biochemical and genetic data suggest that InhA is not the primary target for activated isoniazid in Mycobacterium tuberculosis. [electronic resource] - The Journal of infectious diseases Nov 1996 - 1085-90 p. digital

Publication Type: Journal Article

0022-1899

10.1093/infdis/174.5.1085 doi


Antitubercular Agents--pharmacology
Bacterial Proteins--genetics
Biotransformation
Drug Resistance, Microbial
Isoniazid--metabolism
Lipids--biosynthesis
Mutation
Mycobacterium tuberculosis--drug effects
Oxidoreductases
Promoter Regions, Genetic